Responsibility of Ethics Essay Example for Free

Responsibility of Ethics Essay Ethical standing among community members and business persons is held in high regard.   The matter of trust needs to be had at all times so that people know that their information is safe.   When ethical issues arise, somewhere, somehow there has to be someone who is able and willing to stand up and report the unethical issue.   All professionals are bound by ethics and the responsibility to report wrong doing. In this case, the widow’s attorney is charging the widow 12-14% more than his average percentage with respects to his other clients.    Normal everyday ethics suggest that not only is he acting in bad ethics but obviously is taking advantage of this widow.   Charlie, even if not mandated by law or administrative rule, should inform the widow.   This would be the right and respectful thing to do but is Charlie mandated by any code of ethics to act on this information and inform her is the real question. Charlie, even though he is not the person creating the ethical situation, is bound by ethical codes to inform this widow at the very least but more appropriately the governing agency, of the actions of her attorney.  Ã‚   As stated in the AICPA Code of Professional Conduct for CPA’s â€Å"members should act with integrity, guided by the precept that when members fulfill their responsibility to the public, clients and employers interests are best served.† (ET Section 53 Article II—The Public Interest, ET Section 52 Article I—Responsibilities, ET Section 54 Article III—Integrity    ET Section 56 Article V—Due Care of the AICPA). The biggest ethical issue is honesty, integrity and the personal gain issues.   The attorney stands to gain a substantial amount of money, well more than what he should for his services.   The code of ethics for attorney’s alone is violated is drastic ways.   Some states have statutes on how an attorney charges their client and some rely of good ethical behavior but this aside, attorneys are to charge only what is fair and consistent with how they charge everyone else. This attorney is violating a huge public interest and the widow can file a complaint and the attorney stands to lose his liscense to practice law.   Charlie can also intervene and simply make a phone call to the governing agency as this particular information that he knows, is not bound by the confidentiality laws as this information was derived not by the widows words but by the admittance of the attorney in which there is no confidentiality as there is no client issue due to the fact that Charlie is not asking for advice and the lawyer is not giving it. Works Cited AICPA Code of Professional Conduct.   2006-2008.   http://www.aicpa.org/About/code/sec50.htm New Jersey Judiciary. Office of Attorney Ethics. 2001 http://www.judiciary.state.nj.us/oae/OAEEthicsPamphlet.pdf

Food Poisoning Essay Example for Free

Food Poisoning Essay Introduction: Every year millions of people suffer from food poisoning due to uncontrolled application of agricultural chemicals, environmental contamination, use of illegal additives , microbiological hazards and others but as a result of increasing awareness of consumers and their demands to provide them with safe, wholesome and high quality food have force many food premises to carry out a broad assessment and re-organize their systems of food control in turn to improve efficiency , rationalization of human resources and harmonizing approaches. This assessment of food control system has resulted to shift from the traditional approach which depends mainly on the final product sampling and inspection and move forward toward the implementation of a preventative safety and quality approach based on risk analysis. (FAO, 1998) Risk based approaches has been developed by World health organization (WHO) and Food and Agriculture Organization of the United Nations (FAO) and it is called risk analysis .(WHO,2011)-website Moreover, risk analysis is a combination of three interconnected elements which are Risk management, Risk assessment and Risk communication. (FAO/WHO training manual, 2006) Risk assessment is a scientific evaluation of risk associated with hazard either qualitatively or quantitatively .Risk management is the process where the relevant information of risk including risk assessment results are used to make decisions on how they will control the risk and implementing proper options. Finally risk communication which may define as the process by which exchange of information occur between risk assessors, managers and concerned parties. (http://www.fao.org/docrep/008/ae922e/ae922e04.htm) Government officials: In order to prepare a food products that is safe for human consumption and trade within the country or worldwide as well as maintain public confidence the government officials has a major role in this issue, yet it is clear that the government has no role in the production of food and cannot by itself to make safe or unsafe food. However, the government does play two important roles to reduce the risk of foodborne illness. The first major role is to create food standards and implement it through laws, regulations, inspection, and compliance procedures. These standards range from setting legislation to prevent food adulteration toward defining limits on the levels of pesticides residues as well as levels of pathogenic bacteria, food labelling, preparation and packaging of food. As a consequence, USDAs in recent times implemented HACCP system for meat and poultry to reduce harmful contamination and the risk of food borne illness. The second role is to address food safety problems that are beyond the control of any person participating in the food chain, which require more than a regulatory solution like for example E.coli O157:H7 pathogen which originates in the gut of cattle and is spread through the environment to contaminate water, fresh produce, and beef during the slaughtering process causing a considerable hazard when present in food. Address these and many other food safety problems need a strong research base, and to develop an effective control measures not to mention the cooperation between farmers, livestock producers, food processors, retailers and consumers. Therefore, government officials play a leading role in promoting basic research and cooperation in such cases to reduce the risk of food borne diseases. (Taylor ump; Hoffmann, 2001) In fact, applying risk analysis provides many benefits to all food safety stakeholders including government. In this term it can help food safety regulators to obtain more evidence and information they require to make an effective decision and to identify and apply suitable measures to control the risk in addition to evaluate these measures for example it can be used to assess the maximum level of pesticide residue in food supplies ,increasing the rate of tests ,provide advice to a certain population subgroup, review labelling requirements, and issue a ban and/or recall for the suspected food product in response. Risk analysis can be applied to support and improve food standards such as regulation (EC) no.1829/2003 on genetically modified foods and feed (EU, 2003) and many others standards. It allows the potential costs of compliance for comparison with the expected benefits, and supports setting priorities among the various problems related to food safety. (FAO 87, 2006) In addition to the above advantages, since in article2 (2) from SPS agreement indicates the need of scientific evidence in establishing sanitary and pytosanitary measures. (SPS) though, applying risk analysis will assist the government to meet their commitments and strengthen the basis for the food trade at the international level by increasing access to new market. (FAO 87, 2006) Moreover, gaps and uncertainties in scientific knowledge can be identified using risk analysis approach, which will lead to improve food safety and a better understanding of food related impacts on consumer health. (FAO 87, 2006) In general, risk analysis is used mostly for chemical hazards but recently FAO and WHO expand the scope to cover microbiological hazards as well. (FAO 76) As a result, microbial risk assessment has been used to assess the risk posed by Listeria in a wide range of food commodities and regulatory involvements to control pathogens. (Federal Register / Vol. 66, No. 39 / Tuesday, February 27, 2001 / Proposed Rules) Thus, through risk analysis the government officials can make out opportunities to reduce risk by using it to establish risk based inspection program, development and application of food standards, collaborate efforts with food processors, and consumer awareness. (Taylor ump; Hoffmann, 2001) However, since not all countries have the capabilities and resources to conduct risk assessment ,so they must make use of international data and expertise available besides data that is internationally accepted such as codex standards which accepted as scientifically valid under SPS agreement .(FAO,76) Food processors: On the other hand, food processors as discussed earlier has the main role to produce safe ,wholesome and high quality food to meet customer demands. While world health organisation (WHO) has briefly indicated that food safety is a shared responsibility from farm to fork, http://www.eufic.org/article/en/artid/processing-food-safety-quality-1/ governments and intergovernmental organisation like Codex Alimentarius commission (CAC) has a major responsibility in creating food standards and so it is important to involve food manufacturers in the process of setting standards at the national and international levels. This participation is beneficial to all stakeholders, and government indeed should assist this exchange of information to help ensure supplying safe products. http://www.fao.org/docrep/v2890t/v2890t05.htm Achieving this requires implementing science based systems derived from risk analysis and associated risk management approaches like HACCP for industry. (Hobbs, 2007) However, to carry out a HACCP plan, hazard analysis is required, as it considers as the first of the seven HACCP principles, it is used to assess the probable hazards that could occur through food processing, gathering data regarding those hazards and their severity to the consumer besides the possibility of their occurrence. Once established a hazard analysis using risk analysis, it must be considered throughout the HACCP plan development. (Peter warring book, food quality assurance book) In addition to the above, there are a number of authors who have suggested the use of risk based systems in HACCP plan (Baird-Parker, 1994, 1995;Buchanan, 1995; Notermans and Jouve, 1995; Elliott,1996; Notermans and Mead, 1996). Thus demands to include some aspects of risk into HACCP system are increasing. (T.Mayeres,97) Burden or benefits) (Mayers, 1997) on the other hand stated that the HACCP system is functioning effectively without the need of risk assessment approach, bearing on mind that HACCP has been internationally considered as an effective way to control food borne hazards. However, some elements of risk analysis can be useful in HACCP by increasing the scientific basis of hazard analysis, can help in identification of critical limits, as well as increasing transparency in decision making.( (T.Mayeres,97) Burden or benefits) Part B/i: Owing to the increasing demands of the global market for sea food products, exposure to food borne illness may increase as a consequence. (Book of Iso and HACCP) For instance,

Neurotransmitter Serotonin Cause Depression Psychology Essay

Neurotransmitter Serotonin Cause Depression Psychology Essay According to the World Health Organization, about 121 million people across the globe suffer from depression and the WHO has ranked depression as fourth in a list of most urgent problems worldwide (2). It is the most prevalent psychiatric disorder and is responsible for nearly 850,000 deaths every year. Supporting this fact, statistics have revealed that the use of anti-depressants has soared over 400 percent in the past two decades (3). According to Kresser, a licensed acupuncturist and practitioner of integrative medicine, several chemically distinct anti-depressants marketed under trade names such as Prozac, Zoloft, and Paxil now enjoy immense popularity as anti-depressants (4). Amongst the four classes of anti-depressant medication, namely selective serotonin reuptake inhibitors (SSRIs), atypical depressants, tricyclic anti-depressants (TCAs) and monoamine oxidase inhibitors (MAOIs), SSRIs have been the most widely prescribed medication by physicians. In this essay, the terms S SRI and anti-depressant will be used interchangeably. The current model of SSRIs assumes that a low level of extracellular neurotransmitter, serotonin, is primarily responsible for depression. Serotonin in our body can be found in two places 80 percent of it in our gastrointestinal tract while the rest in our brain (5). The 80 percent of serotonin function as hormones and they play a role in muscular contractions whereas the 20 percent act as a neurotransmitter in our brain (6). In our brain there are many cells called neurons, which are separated by small gaps. Messages carried by neurotransmitters are delivered from one neuron to another across the gaps. These messages come in the form of chemical impulses, and contain information about mood, behaviour, body temperature, appetite and sleep. Once a neurotransmitter leaves the sending neuron, it will latch onto the receiving neuron and relay chemical impulses over. Then the neurotransmitter returns to its sending neuron to be re-used again this process is called reuptake. On the other hand, if there are inadequate amounts of neurotransmitters, the next impulse does not fire off and messages will not be relayed. (7) SSRIs work to block or slow down the reuptake of serotonin in particular, hence increasing the amount of extracellular serotonin. As a result, more serotonin are present in the gaps which will increase rate of successful transmission of impulses to the receiving neuron. SSRI is hence engineered on the belief that serotonin is the cause of depression. However ever since the advent of the drug and its side-effects exposed, drug researchers are compelled to re-investigate the efficacy of SSRIs, in which confounding results were revealed. The investigation into the serotonin-depression link will not only prevent doctors making inappropriate prescriptions that may not be in the best interest of their patients health, it also allows a clearer definition of the causes of depression. Ultimately, establishing the proper function of serotonin may lead to a ground-breaking change in the methodology of treating depression and related disorders in the psychiatric and pharmacology world. While most people concur with the belief that a deficiency of serotonin is related to depression, some argue that an imbalance in serotonin levels leads to depression. This imbalance theory arises because of the widespread notion that SSRIs are only effective for patients with moderate to severe depression while it is ineffective for mildly depressed patients. The basis of the debate surrounding the efficacy of SSRIs in fact boils down to a deeper problem whether or not the neurotransmitter, serotonin, is related to depression at all. Nevertheless, I oppose both claims of the serotonin-depression link and contest that there is no coherence between levels of serotonin and depression. Up till now, there have no substantial evidence that depression is caused by serotonin deficiency, neither is there one that shows that over stimulation of serotonin causes depression. Efficacy of SSRIs challenged by small drug-placebo difference Many studies have shown that the efficacy of SSRI drugs in the treatment of depression is challenged by low drug-placebo difference scores. Studies to investigate the efficacy of anti-depressants by giving placebos to a controlled group have revealed that the recovery rate of patients who took a glucose pill was equivalent to patients who consumed the anti-depression drug (8). A 2008 meta analysis of the efficacy of SSRIs that was published by the Food and Drug Administration (FDA), revealed that these anti-depressants have no clinically significant edge over all placebos. By this, it means that it did not meet the drug licensing authority, UK National Institute of Clinical Excellence (NICE) standards. As evident in the meta analysis, the placebo response groups account for up to 75 percent of all positive effects of anti-depressant medication (9) which shows that 3 in 4 of all patients who reported an increase in heightened emotional well-being were actually consuming sugar pills. O ther studies yielded similar results a study by Khan et al. found a 10 percent difference in level of symptoms when patients consume the inert placebos compared to the active drugs in two separate meta-analyses (10). As the drug-placebo difference is small, it can be seen that regardless of whether SSRI is administered or not, symptoms of depression are still greatly reduced. This implies that serotonin level may not be related to depression at all. Opponents argue that experiments to test the efficacy of SSRIs against inert placebos may not be accurate because the side effects of SSRIs are not mimicked. They claim that commonly known side effects of SSRIs, such as diarrhoea, nausea, dizziness, headaches or even gastrointestinal bleeding (11) may affect patients mood, which in turn underrate the impact of serotonin in lifting depression. This claim is however rejected by many scientific literatures which show counter-evidences. According to Joanna Moncrieff, the co-chair person of Critical Psychiatry Network, when she used active placebos to simulate the adverse-effects of SSRIs in anti-depressant drug trials, results revealed that differences between active placebo and SSRI were significantly small (12). To measure severity of depression before and after the drug trials, the conventional Hamilton Rating Scale of Depression (HRSD) was used. Since it did not meet NICE standards of an improvement in rating score of 3 points to be defined as clinically significant (8), the above studies involving inert and active placebos clearly show that no matter which placebo type was administered active or inert, drug versus placebo significance in anti-depressant efficacy is clinically insignificant. Whether or not the level of serotonin is increased, patients reported a reduction in symptoms of depression, therefore there is little evidence to say that a lack of this neurotransmitter causes depression. Another common belief by proponents of anti-depressants is that initial severity of depression is directly related to the effectiveness of SSRIs, that SSRIs work best for patients with very severe depression. It is thought that over stimulation of serotonin may cause further chemical imbalance in patients suffering from mild depression, hence rendering SSRIs ineffective (13). Thus in order to test this claim, Kirsch et al moved on to investigate whether initial severity of depression affects the efficacy of anti-depressants. He tested on the hypothesis that anti-depressants work only for people suffering from moderate to major depression. In this double-blinded study of 35 clinical trials involving 5,133 subjects, both drug administers and subjects were unknown to results of randomized medication (placebo or SSRI) to prevent sampling biasness and subjects severity of depression was measured by HRSD (14). The test was conducted to see if there is an improvement in the subjects depress ion, measured against their baseline severity and the final conclusion is as follows: patients with an initial moderate depression did not report a drug-placebo difference, patients with an initial severe depression reported a relatively small drug-placebo difference and only for patients situated at the upper end of very severe depression category did the drug-placebo difference fall into the clinically significant criterion by NICE standards (8). Although effectiveness of SSRIs may seem to improve with the severity of depression, further research has revealed a negative coherence between severity and placebo response. As highlighted from Figure 1, the drug-placebo difference reached clinical standards for people with a higher initial severity of depression. Further analysis shows that a higher drug-placebo difference is due to a decrease in improvement of the placebo group rather than due to the effects of SSRIs. Figure 1. Mean Standardized Improvement as a Function of Initial Severity and Treatment Group, Including Only Trials Whose Samples Had High Initial Severity graph.png Source: http://www.plosmedicine.org/article/fetchObject.action?uri=info:doi/10.1371/journal.pmed.0050045.g003representation=PNG_M This implies that the increased benefit for extremely depressed patients seems attributable to a response-deficiency to placebos rather than a heightened response to SSRI medication. Therefore efficacy of SSRI does not increase with severity of depression and increasing amount of serotonin did not work for patients with all levels of depression. Since SSRIs are designed to alleviate depression by inhibiting the reuptake of serotonin in our brain cells, it shows that there is no relationship between extracellular serotonin and ones mood. Furthermore, it usually take weeks before effects of anti-depressants are expressed and can be measured by testing for serotonin levels in the blood, yet patients often report relief within hours or days of medication. Therefore this phenomenon demonstrates the lack of correlation between serotonin and depression and gives support to the placebo effect. The lack in correlation is further evidenced by the results of a Force Swim Test (FST). FST, also known as the behavioural despair test, is a conventional anti-depressant screening test which involves using rodents as test subjects. In this test, rats are dropped in an enclosed water cylinder and their movements observed. The struggling time of rats is measured based on the assumption that immobility of rats is directly proportional to their state of depression. For example depressed rats will cease trying and float in the cylinder, which is akin to despair, whereas non-depressed rats will continue to struggle in search of a way out (15). Although it is thought that SSRIs should extend struggling time of rats, final results were inconsistent hence inconclusive (16). The administration of SSRIs in rodents did not make them less susceptible to depression, displaying no direct relationship between serotonin and depression. Nonetheless, it should be noted that experiments done on mice ma y not be entirely accurate in predicting responses in humans (17). The bold assumption made by researchers The serotonin-depression link came about when scientists first discovered that in most depressed patients, the level of serotonin is comparably lower than that in non-depressed people. The amount of serotonin in a humans body was measured by comparing blood samples taken from depressed and healthy people. Subsequently the anti-depressant SSRI was invented, which targets the neurotransmitter serotonin and works to stimulate the production of it. This methodology then raises a few doubts. Firstly, the assumption that blood serotonin and brain serotonin are directly proportional can be contested as it is certainly impossible to measure the amount of serotonin in the brain. Patients who have high levels of serotonin in the blood may have low levels of serotonin in the brain and vice versa. As mentioned earlier, 80 percent of the humans body total serotonin is found in our bloodstream and the rest in the brain. While the level of blood serotonin can be measured, current biomedical technol ogy has yet to transcend the brain barrier. In all clinical trials involving SSRIs, the assumption made is that blood serotonin reflects brain serotonin, which is a very bold one to make. This then creates a paradox in research methodology: the reason for inventing SSRIs instead of feeding serotonin directly to a humans body is due to the blood-brain barrier. Orally ingested serotonin are ineffective as they do not pass through bloodstreams into the brain, that is the digestive system is unparallel to the central nervous system. Whereas SSRIs work because they merely seek to enhance an impulse that is already present, but too feeble to cross the gap. Yet scientists conveniently established a link between serotonin and depression by measuring serotonin in patients blood. It is reasonable to say that since blood serotonin is not proven to be a clear indication of brain serotonin, any positive outcomes of anti-depressant drug trials may not be due to the increase in brain serotonin bu t other unknown factors. This again shows a lack of tangible evidence between the neurotransmitter, serotonin, and depression. Secondly, in all probability that there is a direct attestation of serotonin deficiency in any mental disorder lacking, it is still unclear whether low levels of serotonin causes depression or depression causes a dip in serotonin. Evidences supporting the latter can be based on observations of non-depressed people with low amounts of serotonin. In a 1996 investigation of the biochemistry of depression, attempts made to induce depression by reducing serotonin levels yielded no consistent results (18). Similarly, researchers found that a surge in brain serotonin, arrived at by administering SSRIs, were ineffective at alleviating depression (19). Therefore there is little evidence to support serotonin as a mood chemical. Also problematic for the serotonin-depression claim is the expanding field of research comparing SSRIs to other anti-depression drugs that do not target serotonin specifically (20). For instance, the atypical anti-depressant buproprion (21) and St. Johns Wort (22), which do not alter the level of serotonin were proven to be just as effective as SSRIs in the treatment for depression. Therefore doubts about the serotonin-depression link are acknowledged by many researchers as well as by advocates of SSRIs (23). To supplement my stand, serotonin is not listed as the cause of depression disorder in the  Diagnostic and Statistical Manual of Mental Disorders (24). The American Psychiatric Press Textbook of Clinical Psychiatry (25) also reiterates that serotonin deficiency as an unconfirmed hypothesis (20). In short, there exists no rigorous corroboration of the serotonin theory, which may suggest the reliability of positive drug trials published by drug companies. Conclusion In addition to what textbooks have to say about serotonin, it is important to look at what not being said in scientific literature. There are numerous peer-reviewed articles supporting the disconnect between serotonin and depression however not a single one can be precisely cited to directly endorse claims of a serotonin deficiency in any mental disorders (20). Assuming that blood serotonin is a good measure for brain serotonin, abundant evidences of high placebo significance in anti-depressant drug trials, the rejection of the claim that efficacy of SSRIs depends on severity of depression, and an inconsistent Force Swim Test results indicate that serotonin may not be the cause of depression. No doubt there may be a positive outcomes from the drug trials, however because blood serotonin may not reflect brain serotonin, these outcomes coupled with the above mentioned proofs against the serotonin hypothesis strongly suggest that other factors are involved in depression. The incongruenc e between the scientific literature and the claims made by proponents are prominent, hence I stand for the fact that there is no direct correlation between serotonin level and depression.

Essay --

In this topic I’m going to analyze quality of life in Dubai.Dubai is one of the most developed city andit is suitable city for life, because everything is high-quality. Also in this essay I will seek to evaluate the positive and negative impacts of quality of life in Dubai, with count: population, social factors, environmental factor, economy factors. Population Actuallynumber of citizen in Dubai is 17% of general population. General population in Dubai is 2.106 million, hence 1.748 million(83%) people are immigrants, such as 52.2% Indian, 13.3% Pakistani, 7.5% Bangladeshi, 2.5% Filipino, 1.5% Sri Lankan, 0.3% American and 5.7% other countries. Everybody knows that UAE is very successful by plenty of oil, free trade and immovable market, but also on using low-paid labor (immigrants) to create city without big expenses.Government understand situation with less number of citizen and with in every way want to increase them and improve life of own citizen. Social Factors: 1)Immigration 1 dollar = 3.67 AED (dirham) As well as known,using immigrants is more cheap than using citizen (i...

Lord of the Flies - Savagery Essay -- English Literature Essays

Lord of the Flies - Savagery â€Å"There are too many people, and too few human beings.† (Robert Zend) Even though there are many people on this planet, there are very few civilized people. Most of them are naturally savaged. In the book, Lord of the Flies, by William Golding, boys are stranded on an island far away, with no connections to the adult world. These children, having no rules, or civilization, have their true nature exposed. Not surprisingly, these children’s nature happens to be savagery. Savagery can clearly be identified in humans when there are no rules, when the right situation arouses, and finally when there is no civilization around us. Without rules, savagery takes over. Without rules, man is free to do whatever he desires. Meaning, their true nature will be exposed. That nature is surely savagery. For example, when you watch little kids, you tend to notice that if one has a toy, the other will start a fight just to get a toy. Since the kids don’t know the difference between rights and wrong, they’re just expressing themselves naturally, which happens to be savagery. Here is a quote from Golding from chapter 4 of his book that proves that rules are the basis to civilization. â€Å"Roger gathered a handful of stones and began to throw them. Yet there was a space round Henry, perhaps six yards in diameter, into which he dare not throw. Here, invisible yet strong, was the taboo of the old life. Round the squatting child was the protection of parents and scho...

20th Century Latin American Literature Essay examples -- Essays Papers

20th Century Latin American Literature Global literatures in English have always played a key role in developing international understanding and appreciation for the social realities and cultural developments beyond Western lifestyles and familiarity. For anthropologists seeking to perceive the social realities of 20th century Latin America, the work of popular authors and novelists of this century is invaluable. Popular authors are the modern mouthpieces of the people and societies who read and love them, and thus, novelists can serve as reliable representatives of the social climate from which they draw material and compose their works. Writers manipulate dominant languages of Latin America â€Å"inherited from rejected colonial powers† to express the content and soul of the people who still live and breathe in these 21 countries of rich heritage and music (Gallagher 1973:1). This collection of countries and peoples has â€Å"experienced parallel histories, cultural formations and literary developments†, and because of these â€Å"profound† ties, constitutes â€Å"a single unit in many senses† (Martinez 1982: 63). Throughout history, â€Å"common Iberian patterns were imposed upon men, cultures and nature that encouraged a blending or unifying process, that is, the creation of the community of nations we call Latin America† and today Latin Americans â€Å"extend their literature in the world†¦no longer concerned about whether or not they express America or their representative countries† (Martinez 1982: 64).However, translating literature from one language to another fo r the benefit of international audiences is a tricky political process, and the history of the evolving Latin American novel testifies to the wild dynamics of language and human identi... ... Texas Press. Martiniez, Jose Luis. Unity and Diversity. In Latin America in Its Culture, vol. 1: Latin America in its Literature. Cesar Fernà ¡ndez Moreno and Julio Ortega, eds. New York: Holmes & Meier Publishers. Minta, Stephen 1987 Gabriel Garcà ­a Marquez: Writer of Colombia. London: Jonathan Cape. Simpkins, Scott. 1995 Sources of Magical Realism/Supplements to Realism in Contemporary Latin American Literature. In Magical Realism: Theory, History Community. Lois Parkinson Zamora and Wendy B. Faris, eds. Durham: Duke University Press. Slemon, Stephen. Magical Realism as Postcolonial Discourse. In Magical Realism: Theory, History Community. Lois Parkinson Zamora and Wendy B. Faris, eds. Durham: Duke University Press. Swanson, Phillip, ed. Introduction: Background to the Boom. In Landmarks in Modern Latin American Fiction. London: Routledge.

Benefits and Features of Excel

Excel is a spreadsheet program. Microsoft Excel is written and distributed by Microsoft. It is used mainly in computers that uses Microsoft Windows operating system or Apple Macintosh computers. It features an intuitive interface and capable calculation and graphing tools. Because of aggressive marketing, Microsoft has made Excel one of the most popular spreadsheet applications that are used these days. It is the dominant spreadsheet application available for Microsoft Windows and Apple Macintosh platforms. The latest releaseof Excel is Microsoft Office 2007 Excel (beta version).Benefits and Features of Excel In Excel user can run queries to sort and filter data, as well as user can run complicated calculations to derive the information they want. One important feature that Excel contains is use of Microsoft PivotTable and Microsoft PivotChart views to work with data interactively. Both uses forms to add, update, delete and navigate user data. Excel is capable of report generations o n data and view in multiple formats. One of the important features Excel contains is to connect to external data and view, query, and edit it without having to import.Excel can imports data form external databases and also capable of export of data too. Excel can create web pages to display data as read-only or to access it in an updatable format and can create a Microsoft Word mail merge (Nelson 2006). Microsoft Excel organizes data in columns. Columns are also called as fields and store a particular kind of information, or data type. At the top of each column, the first cell is used to label the column. Excel is used in the cases when the data is flat. Suppose user has to create a report only once from a set of data, then in this case Excel will be preferred.Second case is when the relationships is not going to change, this means that the structure of the data will remains same so Excel can be used in this case. Another case when Excel can be used is when the report format of the data is not going to change (Kevin 2006). Below is the summary of the cases when the data should be stored in Excel: †¢ If there is a requirement of a flat or non-relational view of data †¢ If the data is mostly numeric. †¢ If user want to run primarily calculations and statistical comparisons on data.†¢ If the dataset is manageable in size (not more than 15,000 rows). (Nelson 2006) References: Nelson, E. 2006. ‘Using Access or Excel to manage your data'. Retrieved on 29 November 2006 from http://office. microsoft. com/en-us/help/HA010429181033. aspx Microsoft Excel. Retrieved on 29 November 2006 from http://en. wikipedia. org/wiki/Microsoft_Excel Kevin 2006. ‘Access Vs. Excel: When to Use Excel'. Retrieved on 29 November 2006 from http://www. workplacelife. com/2006/05/09/access-vs-excel-when-to-use-excel/